Adverse effects of polio vaccines

DDT, Dicofol, infant mortality

I thought that a limited topic like polio vaccines wasn’t too much information, but it looks like there’s a sheer limitless amount of dirt...

In 1972, in the US DDT was prohibited, but instead they continued to produce “dicofol” (a.k.a. Kelthane), which is really DDT in disguise.
K. Paul Stoller – AD, AFP, ALS, and DDT (2015): http://journalijcar.org/sites/default/files/issue-files/0151.pdf
(archived here: http://archive.is/QmJa7)
DDT was/is used in the United States to control insects in crops and livestock and to combat insect-borne diseases. It was introduced as a pesticide during WWII. In the United States, the general use moratorium took place in 1972, but there is another pesticide dicofol, which is made and sold by Dow AgroSciences and carries the trademark KELTHANE® , is in fact DDT. In China dicofol is produced by the Yangzhou Pesticide Factory, which reports production quantities of 4 million pounds of dicofol per year.
(…)
This class of insecticide (organochlorine) impacts the electrical activity in the body, so it affects brain and heart, but also there are other organs that use electrical current, including the lungs. It clearly affects the immune system and causes cancer, and the best part is that it doesn’t break down in the environment other than to become DDE (and DDD).
(…)
India, which manufactures and uses the most DDT, was declared free of polio in 2011, but cases of AFP have skyrocketed.


Stoller refers to a nice paper describing research by the US Army Medical Research and Development Command (with MIT) that shows that at the 20 and 40 μg levels of DDT, the yield of polio virus per cell was increased 37 and 90%, respectively. It is reasonable to postulate that it could also increase the replication of other viruses.
Also look for Kelthane (that replaced DDT) which increases the yield of the polio virus with a whopping 430%...

J. Gabliks – Studies of Biologically Active Agents in Cells and Tissue Cultures (1967): http://www.dtic.mil/dtic/tr/fulltext/u2/804387.pdf
(archived here: http://archive.is/b7CDg)
insecticidal compounds DDT, chlordane, Kelthane , Dipterex malathion, and Karathane at subtoxic concentrations inhibited vaccinia virus replication in human Chang liver cellq. Under the same experimental conditions, the replication of poliovirus was inhibited only by chlordane and malthion, whereas Kelthane increased the virus yields 4 and 18 times, respectivaly, and DDT exhibited a slight stimulatory effect.
(…)
The studies described in the Annual Progress Report of 1965 indicated that human HeLa cells exposed to subtoxic concentrations of the insecticidal compounds, CygonR, DipterexR, DI-SystonR, chlordane and Karathane for 84 days were more susceptible to poliovirus infection than the corresponding control cells (1,2).
As it is recognized that residues of some agricultural insecticides persist in the body and that cells are continuously exposed to their metabolites, we investigated the possibility that these chemicals may alter certain physiological activities of cells and subsequently influence the susceptibility of hosts to virus infections.
The effects of organophosphorus, organochlorine and dinitrophenol insecticidal compounds on the replication of polio and of vaccinia viruses were studied in human Chang liver cells, using purified or analytical grade comnounds obtained from their manufacturers.
(…)
In the poliovirus test the cell response was not uniform. In comparison to the controls, the virus yield in the DDT-treated cultures was slightly increased; the yield in the chlordane and malathion cultures was reduced (32 and 18% of the controls); and the yield in the Kelthane and Karathane cultures was greatly increased (430 and 1800% respectively).
(…)
The stimulatorv effect by DDT, Kelthane, and Karathane on poliovirus replication suggests a possibility that these compounds may have some specific effects on the mechanisms of viral biosynthesis. This possibility is supported by the results of our previous report which indicated an increased yield of poliovirus in HeLa cells exposed to Karathane for 77 days(2).
(…)
Appendix Number 1
EFFECTS OF INSECTICIDAL COMPOUNDS ON THE REPLICATION OF JACCINIA AND POLIO VIRUSES IN HUMAN CHANG LIVER CELLS
Janis Gabliks
(…)
To investiqate the effects of insecticidal comuounds on viral virus replication, we tested six insecticidal compounds in human liver cells infected with vaccinia and poliovirus.
(…)
In the presence of 20 and 40 μg levels of DDT, poliovirus yield per culture was comparable to that of the control. However, when the yield of infectious virus released is expressed per individual cell or per ig of cell protein, as shown in Figure 3, it is evident that at the 20 and 40 μg levels of DDT, the yield of virus per cell was increased 37 and 90%, respectively. Similarly, the yield per μg of protein was also increased 15 and 47%, respectively
(…)
In contrast to the inhibitory action of these two insecticides, the Kelthane-treated cultures produced about four times more virus, and the Karathane-treated cells 18 times more virus than the corresponding controls.
(…)
The stimulatory effect by DDT, Kelthane, and Karathane on poliovirus replication suggests a possibility that these compounds may have some specific effects on the mechanisms of viral biosynthesis, and the state of some latent viruses may also be altered. This possibility is supported by the results of our previous report which indicated an increased yield of poliovirus in HeLa cells, chronically exposed to Karathane for 77 days (7,8).


The following study writes about a previous study, where the Oral Polio Vaccine (OPV) raised the death rate in children (only significant for boys). But this couldn’t be repeated in this study.
This study in Guinea-Bissau is the only in its kind. Can anybody think of any reason for why big pharma wouldn’t research that the polio vaccines causes death…
This study could be completely worthless, as it wasn’t single blind (let alone double blind) and there are also admitted effects that there were other ongoing polio vaccination campaigns, for which the data was manipulated (censored); the study doesn’t describe how it was “censored” (I can’t remember that in the course statistics that I followed at university “censoring” was mentioned...).

Najaaraq Lund et al – The Effect of Oral Polio Vaccine at Birth on Infant Mortality: A Randomized Trial (2015): https://academic.oup.com/cid/article/61/10/1504/302404/The-Effect-of-Oral-Polio-Vaccine-at-Birth-on
(archived here: http://archive.is/LfMUk )
Background. Routine vaccines may have nonspecific effects on mortality. An observational study found that OPV given at birth (OPV0) was associated with increased male infant mortality.
(…)
A supplementation conducted from 2002 to 2004 in Guinea-Bissau, the country experienced a period with a shortage of OPV; a total of 962 trial participants did not receive the recommended OPV0. Surprisingly, boys who missed OPV0 had significantly lower infant mortality compared with boys who received OPV0 (adjusted mortality rate ratio [MRR], 0.35 [95% confidence interval {CI}, .18–.71]) [24]. The tendency was opposite for girls (adjusted MRR, 1.14 [95% CI, .70–1.89]); thus, the effect of OPV0 differed significantly between boys and girls (P = .006). Receiving OPV0 was also associated with reduced immune responses to BCG in both sexes [25].
(...)
Children randomized to OPV received 2 drops of the vaccine orally immediately after randomization. There was no placebo or blinding. Guinea-Bissau does not have a central vaccination registry, so it was important that the vaccination card contained the correct information if a child moved from the HDSS area.
(…)
The main outcome was overall mortality (excluding accidents) within the first 12 months of life. In addition, we analyzed the “pure” effect of OPV0 on survival from enrollment to age 6 weeks, before controls were likely to receive the first routine OPV.
Furthermore, the protocol specified that if large campaigns were conducted during follow-up, analyses would also be conducted with censoring for such campaigns. Thus, we analyzed the impact on infant mortality with censoring for subsequent national OPV campaign, as OPV campaigns could potentially neutralize any differential effect of OPV0. During the conduct of the trial, there was also a national MV campaign (December 2009) and an H1N1 vaccine campaign (October 2010), both targeting children aged 6 months to 5 years. In additional analyses, we censored for these campaigns.
(…)
Based on previous observations [31], we anticipated an infant mortality rate of 50 per 1000 participants. However, during the study period, infant mortality declined to 26 per 1000 person-years. This reduced the power to detect a difference between groups. An interim report for the DSMB showed significantly higher mortality among children randomized to BCG only. As this was in favor of current policy, the DSMB recommended that the number of participants not be increased to compensate for the lower mortality.
(...)
Table 2.
Infant Mortality Rates and Hazard Ratios for Children Randomized to BCG Plus Oral Polio Vaccine at Birth or BCG Only, Guinea-Bissau, 2008–2011
78d514cd308e45730fa21973757a919b9bcc3fff.png

(…)
Within censoring for OPV campaigns (mean duration of follow-up = 163 days), 41 died in the BCG + OPV0 group and 60 died in the BCG-only group.
(...)
Contrary to our initial hypothesis, both male and female children who received OPV0 with BCG tended to have better survival than those who received BCG only. As in a previous study, receiving OPV0 within the first days of life appeared to be associated with the strongest benefits. Many OPV campaigns occurred during the trial; when censoring for these campaigns, the beneficial effect of receiving OPV0 was borderline significant overall and significant separately in males.
(...)
A weakness of our study is that it was not blinded. However, health workers in the area were unaware of the study hypothesis and were unlikely to be influenced when treating the children. The outcome assessment was carried out by assistants unaware of the vaccination allocation. Hence, we do not believe that the lack of blinding affected the results.
In addition, national OPV campaigns could have led to environmental acquisition in children born in the period after a campaign. It is likely that, in a population-dense urban setting, indirect vaccination also occurs in the absence of OPV campaigns, through close contact with newly vaccinated children. This exposure, if anything, would be expected to dilute any differences between children who received OPV0 or no OPV0.
All background factors were evenly distributed between the 2 randomization groups and did not confound the results. OPV0 had the best effect among children enrolled early. Early enrollment could be a proxy for better socioeconomic status, as it is typically the more wealthy mothers who delivered at the maternity ward and were enrolled early.
(...)
In 2004 there were several campaigns, and none in 2007, but many during the present trial. Censoring for these campaigns made the results more comparable, as OPV0 tended to be most beneficial before additional OPV was given in campaigns.


Also see the rising numbers in reported AFP cases from 1993 to 1995 in Cambodia, China, Lao PDR, Philippines, in the 6 countries totally from 2674 to 5644 (more than double in 2 years).
Rudolf H. Tangerman et al – Poliomyelitis Eradication in the Western Pacific Region (1997):
https://www.google.nl/url?sa=t&rct=...ll.pdf&usg=AFQjCNEa_S8s7B7lQ7QiTcPDCSM3hhCOWA
(archived here: http://archive.is/mhrzS)
5bc44d92b69654ddc826264a847d0c28d27e6a4a.png
 
Last edited:
Infant Mortality

Firestarter said:
The following study writes about a previous study, where the Oral Polio Vaccine (OPV) raised the death rate in children (only significant for boys). But this couldn’t be repeated in this study.
Benn et al – Sex-Differential Effect on Infant Mortality of Oral Polio Vaccine Administered with BCG at Birth in Guinea-Bissau... (2008): http://journals.plos.org/plosone/article?id=10.1371/journal.pone.0004056
(arvchived here: http://archive.is/j3DKR)
The effect of OPV at birth on overall child mortality was never studied. During a trial of vitamin A supplementation (VAS) at birth in Guinea-Bissau, OPV was not available during several periods. We took advantage of this “natural experiment” to test the effect on mortality of receiving OPV at birth.
(...)
A total of 962 (22.1%) of the 4345 enrolled children did not receive OPV at birth; 179 children died within the first year of life. Missing OPV at birth was associated with a tendency for decreased mortality (adjusted MRR = 0.69 (95% CI = 0.46–1.03)), the effect being similar among recipients of VAS and placebo. There was a highly significant interaction between OPV at birth and sex (p = 0.006). Not receiving OPV at birth was associated with a weak tendency for increased mortality in girls (1.14 (0.70–1.89)) but significantly decreased mortality in boys (0.35 (0.18–0.71)).
(...)
Furthermore, we also conducted one study of an OPV campaign; receiving OPV in the campaign compared with not receiving OPV was associated with significantly decreased mortality among the youngest children [9].
(...)
A total of 962 children (22.1%) of the 4345 enrolled children did not receive OPV at birth (Appendix S1). There were significant differences between those who did and those who did not receive OPV at birth (Table 1). Not having received OPV at birth was associated with low age at enrolment, no maternal school education, low arm circumference, being enrolled at the Belem Health Centre, and dry season (Table 1).
(...)
The cumulative mortality curves for all children and for boys and girls separately, are illustrated in Figures 2A–C. The effect of not receiving OPV at birth on mortality up to 2 months of age (53 deaths) was 0.39 (0.12–1.29) among boys and 1.38 (0.54–3.57) among girls (p = 0.099). However, the effect of not receiving OPV at birth was not limited to the first months of life, but appeared to continue throughout the first year of life.
534d0f4ebe33daad0cd185e8aa56231cfb51672e.png

(...)
We studied the effect of missing OPV at birth on the major causes of death: malaria, diarrhoea, respiratory diseases and septicaemia; 58 deaths had other less common causes of death or had no verbal autopsy conducted because the family had moved. Though none of the differences reached statistical significance, the sex-differential effect of OPV at birth was seen for all four major causes of death (Table 3).
(...)
Among study children, a total of 366 hospitalisations took place from enrolment to 12 months of age. We found no effect of missing OPV at birth on the risk of hospitalisations within the first year in either sex; the adjusted IRR among boys being 0.84 (0.59–1.20) and 1.05 (0.68–1.63) among girls. The effect of missing OPV at birth on hospital case-fatality also did not differ between boys and girls, the adjusted relative risks being 1.04 (0.45–2.40) and 1.30 (0.55–3.07), respectively.
(…)
It strengthens the observation that the effect of not receiving OPV was comparable during the two major independent periods without OPV. Though no significant associations were observed between not receiving OPV at birth and major causes of death, hospitalization or hospital case-fatality, the pattern was the same. For all major causes of death there was lower mortality for boys and higher for girls if they had not received OPV; for both hospitalization and case-fatality there was a 20–25% difference between boys and girls which went in the same direction as the overall result. Hence, the results are compatible with the hypothesis that OPV at birth increases mortality of infant boys. Many factors influence a mother's choice to bring the child to the hospital, and hospitalizations may not be the best proxy for severe disease. The vast majority of children died outside the hospital.
 
Vitamins B1, C, Calcium, Sugar

I finally found something positive to write about polio prevention...
Generally speaking you could prevent polio paralysis, by: getting enough vitamin B1 (thiamine), vitamin C, Calcium and cut the intake of sugar.
There is only information on this in “scientific” papers going back to the 1950s or even before. Why would the pharmaceutical industry stop the research into prevention of paralysis by other methods than vaccines that cause more harm than they do good?
It’s not sure however that what worked, also has beneficial effects with all of the pesticides used today, some even worse than DDT.


MCCORMICK (1938)
McCormick’s report from 1938 shows that vitamin B deficiency possibly increases the susceptibility for polio, and that Adrenalin, Cortin and Vitamin C destroy the polio virus. It is a pretty good literature review.
W. J. MCCORMICK – POLIOMYELITIS: VITAMIN B DEFICIENCY A POSSIBLE FACTOR IN SUSCEPTIBILITY (1938): https://www.ncbi.nlm.nih.gov/pmc/articles/PMC536391/pdf/canmedaj00190-0052.pdf
Zingher, in a report on over one thousand cases of poliomyelitis under 5 years of age, found over 80 per cent to be Schick-positive. Baginsky says: "I have been struck with the very extraordinary sensitivity of children suffering with such diseases of the central nervous system as poliomyelitis, spastic cerebrospinal paralysis, hemiplegia, etc. One is scarcely able to keep these children in the hospital wards free from infection with diphtheria." The situation in this respect at the present time may be somewhat altered owing to the general adoption of toxoid immunization.
In a comparative study of poliomyelitis and diphtheria Jungeblut4 reports poliocidal substances occasionally demonstrable in the serum of monkeys actively immunized against diphtheria toxin. He and his associates also found that contact in vitro with adrenalin, cortin and vitamin C is equally destructive for both the toxin of diphtheria and the virus of poliomyelitis.
(…)
On the assumption of possible exogenous factors in susceptibility, based on nutritional imbalance, the recent work of Jungeblut and Toomey is of interest. The former, in experimental poliomyelitis in monkeys, has obtained a fair degree of protective effect from vitamin C when administered within a rather definite range of dosage. The latter has demonstrated increased susceptibility when vitamin D is withheld in conjunction with a diet otherwise rich in vitamins. But, strange to say, little or no work of a conclusive nature has been done in this respect with the anti-neuritic vitamin B. With the established anti-paralytic effect of vitamin B in beriberi it would seem that this pioneer in the vitamin field should offer the most hope of protective value in poliomyelitis.
(…)
Based on the obvious assumption of a vitamin B deficiency, a radical change in the native dietary was instituted. The sale of white flour and polished rice was forbidden in the native stores, whole-meal flour and brown rice being substituted, and the use of whole wheat bread was encouraged. Sweet potatoes were introduced, and the value of eggs and condensed milk was stressed. The sale of sugar was restricted to 1 lb. per adult per week. The use of the yeast-laden cocoanut-sap beverage was again permitted, and the sick babies were treated with an emulsion of the yeast in cod-liver oil. As a result of these measures the infant death rate rapidly fell from 50 to 7 per cent, at which level it has consistently remained.


JUNGEBLUT (1936 & 1939)
McCormick references an earlier paper by Jungeblut.
I have found 2 of his papers documenting research on the effects of vitamin C in monkeys infected with polio. The results appear to indicate that vitamin C has positive effects in fighting polio.

CLAUS W. JUNGEBLUT – VITAMIN C THERAPY AND PROPHYLAXIS IN EXPERIMENTAL POLIOMYELITIS (1936): http://jem.rupress.org/content/jem/65/1/127.full.pdf
The limited data which formed the basis of this preliminary report naturally precluded the drawing of any definite conclusions. They seemed to suggest, however, that vitamin C when administered in the proper dose may possess distinct therapeutic properties in experimental poliomyelitis.
(…)
4. A summary of the results obtained in all three groups shows: (a) that among a total of 62 treated monkeys, 19 survived without paralysis and 43 succumbed to the disease, while of a total of 38 untreated controls, only 2 failed to develop paralysis and 36 succumbed to the disease; (b) that treatment with large doses of vitamin C was without any beneficial effect (all 10 monkeys which had received 700 to 100 rag. developing paralysis), that the administration of intermediate doses was followed by occasional survival without paralysis of the treated animal (3 monkeys surviving of a total of 19 which had received 50 to 10 rag.), and that nearly one-half of the animals which had received small doses escaped the disease (16 monkeys surviving of a total of 33 which had received 5 rag.).

In the 1939 report, Jungeblut describes a range of experiments with vitamin C and the polio virus, again in monkeys.
CLAUS W, JUNGEBLUT - A FURTHER CONTRIBUTION TO VITAMIN C THERAPY IN EXPERIMENTAL POLIOMYELITIS (1939): https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2133810/pdf/315.pdf
Since the very beginning of this investigation it had been realized that the measure of therapeutic success attainable by vitamin C administration depended upon certain variables the operation of which was not clearly understood. Most important among these was the kind and dosage of vitamin C, optimal results being obtained only with optimal doses of natural vitamin C.
(…)
It will be seen from Table II that all of the monkeys receiving the vitamin C-virus mixtures remained free from paralytic symptoms while both control monkeys injected with the virus-distilled water mixtures developed complete paralysis on the 7th and 8th day, respectively. This experiment therefore confirms our previous observation that small amounts of vitamin C suffice to inactivate multiple paralytic doses of poliomyelitis virus in vitro.
(…)
Somewhat different results were obtained in the three series with a less drastic mode of infection. Not only was the disease produced by the droplet method of instillation much less severe than that induced by the pressure method, but the symptoms became progressively lighter from series to series in both groups of animals, treated and untreated. Undoubtedly, the droplet instillation created conditions under which only minute amounts of virus reached the olfactory area, allowing for a variable number of animals to develop typical paralysis of the extremities while the remainder showed either slight transitory involvement of the central nervous system or escaped paralysis entirely.
TABLE IV Effect of Vitamin C Treatment in Monkeys Infected Intranasally
4d801488239c8eff6c1504a3bed0c325ce375226.png

(…)
The results recorded in this paper serve to affirm and extend our previous observations on the effect of crystalline vitamin C on poliomyelitis virus. They clearly show that small amounts of ascorbic acid, either natural or synthetic, are capable of inactivating in vivro multiple paralytic doses of two different strains of this virus. This virucidal action of vitamin C, however, is by no means limited to poliomyelitis virus.
(…)
With a less forceful method of droplet instillation, the picture of the disease in control animals becomes so variable that the results cannot be easily interpreted; but the available data suggest that vitamin C treatment may be a factor in converting abortive attacks into an altogether non-paralytic infection. 3. The administration of synthetic vitamin C to monkeys infected intracerebrally with small doses of the RMV strain gives results comparable to those previously obtained with this substance in monkeys infected intracerebrally with the Aycock strain of poliomyelitis virus.

Jungeblut found out that a therapy with vitamin C only works if it’s not too low or too high. Jungeblut also discovered that if the polio virus is given too abundantly, the vitamin C therapy doesn’t work. It’s obvious that in a “normal” situation somebody gets infected with only a limited amount of the polio virus.
Knowing this, the pharmaceutical industry can easily manipulate a “scientific” trial to “prove” that vitamin C is useless for a polio therapy…


KLENNER (1949 & 1951)
Fred R. Klenner was a doctor who experimented with vitamin C treatment for polio and other viral diseases, but unfortunately from a “scientific” point of view his work is worthless.
Here’s one of his papers from 1949, where he claims that vitamin C cured all of his polio patients in a couple of days: https://www.vitamincfoundation.org/www.orthomed.com/polio.htm

Klenner’s paper from 1951 is better. The problem is that Klenner apparently developed a therapy for his patients, but without a control (placebo) group this is “scientifically” worthless: http://www.seanet.com/~alexs/ascorbate/195x/klenner-fr-southern_med_surg-1951-v103-n4-p101.htm
It is of more than academic interest to review the findings of McCormick in 50 confirmed cases of poliomyelitis in and around Toronto, Canada, during the epidemic of 1949. This report is that children of families eating brown bread who came down with poliomyelitis did not develop paralysis; whereas in those families eating white bread many of the children having poliomyelitis did develop paralysis. The point here is that brown bread has 28 times more vitamin B[SUB]1[/SUB] than does white bread. Obviously, then, the paralysis which complicates acute poliomyelitis appears to be due to a B[SUB]1[/SUB] avitaminosis. Vitamin C by removing edema fluid relieves from pressure these vessels that supply nutriment to the horn cells, thus allowing the normal complement of vitamin B[SUB]1[/SUB] to reach these cells.
(…)
After 96 hours the child was moving both legs. The flexion was slow and deliberate. She was discharged from the hospital at this time, vitamin C being continued by mouth — 1000 mg. every two hours with fruit juice for seven days. On the 11th day of treatment the child was walking about the house, but her gait was slow and her posture was poor, being bent forward. Vitamin C was discontinued and vitamin B[SUB]1[/SUB] started — 10 mg. before meals and bed hour. Carbonated drinks were encouraged for their sugar content and mild stimulating action. Nineteen days after starting treatment there was complete return of sensory and motor function which has persisted to this date.
(…)
Calcium, in vivo, duplicates the chemical behavior of vitamin C in many respects. Calcium gluconate and calcium lexulinate were used in conjunction with vitamin C therapy in a small series of pulmonary virus infections and in mild cases of influenza.


THE VITAMIN COVER-UP – JONAS SALK
Jonas Salk is known for creating the first polio vaccine; coincidentally it was the same Salk who was involved with the cover-up of the research of Claus Jungeblut.

On 18 September 1939, Time magazine reported that Jungeblut, showed the correlation between vitamin C deficiency and polio.
Within 2 years after the discovery of vitamin C (a.k.a. ascorbic acid), Jungeblut showed that ascorbic acid could inactivate the polio virus.
In 1936-1937, there was a rapid succession of medical scientists, who showed that vitamin C inactivated a variety of viruses: Holden et al (herpes); Kligler and Bernkopf (vaccina virus); Lagenbusch and Enderling (hoof-and-mouth disease); Amato (rabies); Lominski (bacteriophage); and Lojkin and Martin (tobacco mosaic disease).
Thus, already in the 1930s it was known that vitamin C was potentially a wide-spectrum antiviral agent.
It was none other than Dr. Albert B. Sabin, who in 1939 published a paper that demonstrated that vitamin C had no value in combating polio viruses. His 1939 paper showed that vitamin C had no effect in preventing paralysis in rhesus monkeys infected with polio.
Klenner said that there was a simple reason for Sabin's well-reported failures: the dosage was far too low: http://orthomolecular.org/resources/omns/v09n16.shtml
(archived here: http://archive.is/QaN2C)


Maybe I will post about the effects of sugar on polio at another time…
 
Last edited:
Propaganda was used heavily to promote the National Foundation for Infantile Paralysis and the March of Dimes solution to the polio epidemics - a vaccine.

This clip is just a small taste of how American citizens were blanketed with propaganda.

 
Diet prevents polio

I had read about the hypothesis that a low intake of sugar prevents polio, based on the story of Benjamin P. Sandler, who changed the diet in the town of Asheville and the surrounding places, which reduced the number of polio patients.
Sandler had a one-track mind and was focused solely on preventing a low sugar level in the blood. I don’t think that Sandler should be taken too seriously, but it certainly is an interesting story.

Here’s Sandler’s– Diet Prevents Polio(1951): http://www.whale.to/a/sandler_b.html
(PDF version: https://www.seleneriverpress.com/images/pdfs/DIET PREVENTS POLIO.PDF)
One of my problems with this paper that the information is displayed very inefficiently, blowing it up to 50 pages...

On 4 August 1948, the Asheville Times, published an article claiming that a change in diet would prevent polio (at the time, because of all the propaganda, the US population was terrified of polio). An important part of the advised diet was a reduction in sugar (which ironically, according to Sandler, prevents a low sugar level in the blood).
Store sales of sugar, candy, ice cream, cakes, soft drinks, dropped sharply and remained at low level for the rest of the summer. One southern producer of ice cream shipped one million fewer gallons of ice cream than usual. The results were impressive, see how the rise in number of polio immediately stopped after the publication.
fig8%20jpg.jpg


Until 4 August 1948, Asheville had 55 cases of polio (more than in 1946 in the same period). For the rest of the year (December 31) there were only 21 new cases, 76 cases for the entire season.
I’m not sure about the plotted estimates for 1948, but the number of polio cases was certainly lower than would have been estimated starting on 4 August, as the result of the change in diet.
Strangely missing are other years. For example 1947…
According to Sandler the success of the diet was even greater in 1949, with only 5 cases. He could have presented this information much better. And why no information on 1950?

This was the published diet advice on 4 August 1948:
(1) Eliminate from the diet sugar and foods containing sugar, such as: soft drinks; fruit juices (except tomato juice); ice cream; cakes, pastries, pies; candies; canned and preserved fruits. (Saccharin may be substituted for sugar.)
(2) Cut down the consumption of starchy foods, such as: bread, rolls, pancakes; potatoes; rice; corn; cereals and grits.
(3) Substitute for such starch foods and starchy vegetables, the following: tomatoes, string beans, cucumbers, greens, lettuce, turnips, carrots, red beets, cabbage, onions and soybeans.
(4) Do not eat fresh fruits or melons more than once a day, and then only in small quantities.
(5) Eat more protective protein foods, such as: eggs, pork and beef products; fish (fresh or canned); poultry; milk, cream and cheese.

When I read this list, the change in diet isn’t limited to sugar…
Maybe this diet increased the intake of vitamin B1?
What’s also notable is the increase in “protein food”, meat, fish and animal products. We are told that we should reduce the intake of “animal” food and go vegetarian, but from the point of view of health this is probably not so good.

Because Sandler was obsessed with preventing a low blood sugar level he even advised to limit “fresh fruits”.
Sandler also advised against sports with a high intensity, as these cause a low sugar level.

Also interesting is the following quote from the paper, which indicates that there were more polio cases on farms, which contradicts that “polio” is caused by a virus. The farms are of course sprayed with pesticides…
If isolation had any effect, it should have been manifest in the county, which is largely rural with widely separated homes. Many of the county cases came from farms. Lack of contact was thus no protection against the disease.
 
Last edited:
The rest of the US which did not go on the diet plan saw a similar change in polio numbers.

polio_cases.jpg


India saw their cased drop from about 30,000 a year to practically zero in a short period of time. Was that because the entire country was eating better?
 
You're not really insinuating that India is a great success of the polio eradication campaign, are you?

India was declared free of the wild polio virus by the WHO in January 2011 however there has been a huge increase in cases of NPAFP.
In 2004, 12,000 cases of NPAFP were reported which increased to 25,000 in 2005, 40,000 in 2007, 61,000 in 2011, and 53,563 cases by 2012.
Government surveillance data show that India has become the nation with the world’s highest rate of NPAFP incidence. In the 13 months before December 2015, India has reported 53,563 cases of NPAFP at a national rate of 12 per 100,000 children: http://www.oye.news/news/health/ind...s-non-polio-paralysis-increased-dramatically/
(archived here: http://archive.is/NM0Tp)



The best source I’ve found on this black page in history are Neetu Vashisht and Jacob Puliyel of the Department of Pediatrics at St. Stephens Hospital in Delhi.

Neetu Vashisht et al – ”Polio programme: let us declare victory and move on” (2012): http://www.whale.to/v/polioaccuteflacid.pdf
(archived here: http://archive.is/3gGM5)



Neetu Vashisht et al – “Trends in Nonpolio Acute Flaccid Paralysis Incidence in India 2000 to 2013” (2015): http://www.ineip.org/content/abstract/764/pp037-14-trends-non-polio-acute-flaccid
(archived here: http://archive.is/GsCls)


The following graph shows that the correlation between number of NPAFP cases and Cumulative amount of Oral Polio Vaccines
3_4.png
 
I try hard to stick to the facts but I'm going to make an exception.

You should be banned for trolling. Polio isn't NPAFP. Nobody here has denied there's something odd going on in those provinces (but only those provinces). But you ignore those statements and fill the forum with the same garbage over and over.

On other topics you have literally posted comments indicating you don't believe science to be true, even though all the evidence indicates it is. You actually post articles, adding long commentary like "I know this says this, but I don't believe it. But I can't supply any evidence to prove otherwise." That isn't discussion - that's propaganda at best.

You don't ever post anything political. These are political forums. You simply do not belong here. I suspect you're only here because other forums have already grown weary of your shenanigans.
 
Last edited:
You're the one trolling this forum with state propaganda over and over again.
This subforum is about "Health care".
In our Brave new world, science is about as dead as independent journalists...

I replied to Zippyjuan, who like you, appears to ignore the facts that were at first paralysis cases were labelled as polio, after the introduction of "vaccines", they were simply labelled as NPAFP, while the cases of paralysis skyrocketted (especially in the third world).

This looks like a political topics to me by the way: http://www.ronpaulforums.com/showthread.php?513623-Michael-Flynn-Inovo-and-Erdogan
 
Last edited:
I replied to Zippyjuan, who like you, appears to ignore the facts that were at first paralysis cases were labelled as polio, after the introduction of "vaccines", they were simply labelled as NPAFP, while the cases of paralysis skyrocketted (especially in the third world).

That's just a flat out lie.

Acute flaccid paralysis is a symptom of polio, and is a symptom that health workers look for when looking for polio outbreaks. But other viruses cause it as well.

Also like I said above - there are two provinces in India where NPAFP did indeed skyrocket after receiving a specific type of polio vaccine. But because that does not happen globally, nobody knows why that happened there. But you'll no doubt ignore that again as well.

This is precisely why I am accusing you of trolling. All of this has been addressed previously but you ignore it and then post it again later.
 
Last edited:
How the heck did 'Lamp' get full green bars already? Who's repping that troll like that?

Prepare for my .50 neg, 'Lamp'. And in succession. Ducking won't work, you'll still catch a bunch of .22 negs even if you duck.
 
Last edited:
That's just a flat out lie.

Acute flaccid paralysis is a symptom of polio, and is a symptom that health workers look for when looking for polio outbreaks. But other viruses cause it as well.

Also like I said above - there are two provinces in India where NPAFP did indeed skyrocket after receiving a specific type of polio vaccine. But because that does not happen globally, nobody knows why that happened there. But you'll no doubt ignore that again as well.

This is precisely why I am accusing you of trolling. All of this has been addressed previously but you ignore it and then post it again later.

There are many causes. https://en.wikipedia.org/wiki/Flaccid_paralysis

This abnormal condition may be caused by disease or by trauma affecting the nerves associated with the involved muscles. For example, if the somatic nerves to a skeletal muscle are severed, then the muscle will exhibit flaccid paralysis. When muscles enter this state, they become limp and cannot contract. This condition can become fatal if it affects the respiratory muscles, posing the threat of suffocation.[2]

AFP is also associated with a number of other pathogenic agents including enteroviruses, echoviruses, West Nile virus, and adenoviruses, among others.

Botulism can also cause it.
 
Firestarter said:
I replied to Zippyjuan, who like you, appears to ignore the facts that were at first paralysis cases were labelled as polio, after the introduction of "vaccines", they were simply labelled as NPAFP, while the cases of paralysis skyrocketted (especially in the third world).
That's just a flat out lie.

Acute flaccid paralysis is a symptom of polio, and is a symptom that health workers look for when looking for polio outbreaks. But other viruses cause it as well.
You're the one lying and attacking any forum member ad hominem that doesn't agree with the state propaganda.
I haven't even claimed that polio never causes paralysis, but that is surely possible based on the evidence I've seen.

You keep on attacking me with your troll posts, without any evidence at all.
And Zippyjuan claimed that India is a great success for the polio vaccines, while the rate of paralysis has skyrocketed, which I replied to with information that's already in this thread.

You and Zippyjuan just keep on ignoring the fact that after the introduction of polio vaccines, the definition for the polio diagnosis was simply changed, which doesn't mean that the polio cases actually went down after the introcution of the polio vaccines, see the following quote:
Donnay said:
11. In 1956 the AMA (The American Medical Association) instructed each licensed medical doctor that they could no longer classify polio as polio, or their license to practice would be terminated. Any paralysis was now to be diagnosed as AFP (acute flaccid paralysis) MS, MD, Bell's Palsy, cerebral palsy, ALS (Lou Gehrig's Disease), Guillian-Barre etc etc.
 
BOMBSHELL: United Nations admits latest outbreak of polio in Syria was caused by polio vaccines

In war-torn Syria, there’s little doubt that the conflict has taken a toll on health. Now, the United Nations Children’s Fund (UNICEF) reports that their attempt at using vaccines to “protect” children against polio has backfire — infecting more children with the devastating disease rather than saving them.

Vaccines make children sick, you don’t say?

As World Health Organization representative Elizabeth Hoff reports, “As of 18 August 2017, 33 children under the age of five have been paralyzed. The detection of the circulating vaccine derived polio virus type 2 (cVDPV2) cases demonstrates that disease surveillance systems are functional in Syria. Our priority now is to achieve the highest possible polio immunization coverage to stop the circulation of virus.”

Thirty-three children paralyzed by the vaccine that was supposed to protect them — and the UN’s response is to just keep doling out more vaccines? Sadly, that’s par for the course. Obviously, bad medicine is never the problem — there’s just not enough bad medicine, that’s all.

Fran Equiza, a UNICEF representative commented, “No child should have to live with devastating effects of polio.” And yet, this is the second outbreak of polio to strike Syria since the conflict first took roots in 2011.

Earlier this summer, NPR reported on this very phenomenon, wherein mutant strains from the polio vaccine caused more paralysis than wild polio. Could it really be that a lab-altered version of a virus is more dangerous than the one found in nature? Absolutely.

As Jason Beaubien reports, as of June 2017, there were more cases of child paralysis caused by the polio vaccine than the actual, wild-caught disease itself. At the time of his reporting, just six cases of “wild” polio, which is naturally occurring in the environment, had been reported worldwide. Conversely, 21 cases of vaccine-derived polio had been reported internationally in June. Obviously, in Syria that number skyrocketed over the last two months — 33 children in Syria alone have been paralyzed by vaccine-derived polio.

Raul Andino, a professor of microbiology at the University of California at San Francisco explains, “We discovered there’s only a few [mutations] that have to happen and they happen rather quickly in the first month or two post-vaccination. As the virus starts circulating in the community, it acquires further mutations that make it basically indistinguishable from the wild-type virus. It’s polio in terms of virulence and in terms of how the virus spreads.”

Andino’s research also shows that the polio virus used in the vaccine will replicate inside human hosts’ intestines. In places with poor sanitation, this often means it has the chance to spread from person-to-person with relative ease. Places where conflict and war have decimated health care systems are known for being breeding grounds for disease outbreak.

It comes as no surprise (sadly) that war-weary Syria is expected to have even more cases of polio in the coming months. Michael Zaffran, the director of polio eradication at the World Health Organization contends, “In Syria, there may be more cases coming up.” Zaffran adds that the WHO is aware of the risk that comes with the live polio vaccine and says it is “a very regrettable hiccup for the poor children that have been paralyzed, of course. But with regards to the whole initiative, you know it’s not something that is unexpected.”

He added that while the children’s paralysis is unfortunate, “from a public health perspective, the benefits far outweigh the risk.” Do you think that will comfort the children and their families as they struggle with the after-effects of vaccine-derived polio? [Related: Learn more about the risks of inoculation at Vaccines.news]

Sources for this article include:

UNICEF.org.hk

UN.org

NPR.org
https://www.vaccines.news/2017-08-3...io-in-syria-was-caused-by-polio-vaccines.html
 
You have 7 negative (red) reputation bars in the your profile. Count 'em. SEVEN! Why would anybody listen to you? People here are either well aware of what Ron Paul says or can easily find it. Your contrarian nonsense is a detriment.




+ rep

You have a bit of Zippy derangement syndrome here, my friend.

Ron Paul is indeed inconsistent on the issue. He stands where I do - on the side of science and freedom. He says vaccine mandates are dangerous, not that vaccines are dangerous.

If I were still a practicing ob-gyn and one of my patients said she was not going to vaccinate her child, I might try to persuade her to change her mind. But, if I were unsuccessful, I would respect her decision. I certainly would not lobby the government to pass a law mandating that children be vaccinated even if the children’s parents object. Sadly, the recent panic over the outbreak of measles has led many Americans, including some self-styled libertarians, to call for giving government new powers to force all children to be vaccinated.
 
I haven't even claimed that polio never causes paralysis, but that is surely possible based on the evidence I've seen.

:

You claimed that the only reason that polio cases dropped is because they renamed poio NPAFP. That is a lie. Then you said the rate of paralysis skyrocketed across India as the polio vaccine was introduced. We pointed out that was also incorrect based even on the data you presented.


The first time you said those things we told you you were wrong and showed you why. Now that you continue to say them is evidence that you're just a liar and/or a troll.

The only reason you see no evidence because you're not actually a researcher. Duh.

As for Donnay's quote, she didn't research her source. She just quoted someone who is wrong. Par for the course.
All you have to do is look around. You don't know anybody with polio. You don't live in fear of catching in. Nobody in your family is in an iron lung. 70 years ago at least one of those statements would be untrue in America. 15 years ago it would still be true in India.
 
Last edited:
The nonsense by Angelatc just keeps on coming
Mark my words- these freaking people are as politically toxic as the white supremacists in the real world.

Now why does Angelatc keeps on posting trolling posts in the health section of this forum?!?
You don't ever post anything political. These are political forums. You simply do not belong here. I suspect you're only here because other forums have already grown weary of your shenanigans.


You claimed that the only reason that polio cases dropped is because they renamed poio NPAFP. That is a lie.
That is not a lie, I'll repeat the relevant quote again...
Donnay said:
11. In 1956 the AMA (The American Medical Association) instructed each licensed medical doctor that they could no longer classify polio as polio, or their license to practice would be terminated. Any paralysis was now to be diagnosed as AFP (acute flaccid paralysis) MS, MD, Bell's Palsy, cerebral palsy, ALS (Lou Gehrig's Disease), Guillian-Barre etc etc.


He stands where I do - on the side of science and freedom.
Nope, you stand on the side of state propaganda and censorship...
 
Last edited:
Back
Top